Home
About
Overview
Sharing Data
ORCID
Help
History (2)
MYB-QKI rearrangements in angiocentric glioma drive tumorigenicity through a tripartite mechanism.
Towards a fully-synthetic substitute of alginate: development of a new process using thermal gelation and chemical cross-linking.
See All 2 Pages
Find People
Find Everything
Login
to edit your profile (add a photo, awards, links to other websites, etc.)
Edit My Profile
My Person List (
0
)
Return to Top
MYB-QKI rearrangements in angiocentric glioma drive tumorigenicity through a tripartite mechanism.
MYB-QKI rearrangements in angiocentric glioma drive tumorigenicity through a tripartite mechanism. Nat Genet. 2016 Mar; 48(3):273-82.
View in:
PubMed
subject areas
Carcinogenesis
Cell Line, Tumor
Child
Comparative Genomic Hybridization
Exome
Gene Expression Regulation, Neoplastic
Gene Rearrangement
Glioma
High-Throughput Nucleotide Sequencing
Humans
Mutation
Oncogene Proteins v-myb
Oncogene Proteins, Fusion
RNA-Binding Proteins
authors with profiles
Peleg Horowitz