Home
About
Overview
Sharing Data
ORCID
Help
History (64)
Developmental mechanisms of human congenital heart disease
Flagellin peptide flg22 gains access to long-distance trafficking in Arabidopsis via its receptor, FLS2.
Goals and targets for personalized therapy for HCC.
Diphenylcarbazide
Modulating vascular intimal hyperplasia using HSV-1 mutant requires activated MEK.
See All 64 Pages
Find People
Find Everything
Login
to edit your profile (add a photo, awards, links to other websites, etc.)
Edit My Profile
My Person List (
0
)
Return to Top
Modulating vascular intimal hyperplasia using HSV-1 mutant requires activated MEK.
Modulating vascular intimal hyperplasia using HSV-1 mutant requires activated MEK. Gene Ther. 2013 Feb; 20(2):215-24.
View in:
PubMed
subject areas
Animals
Carotid Arteries
Cell Line
Cell Proliferation
Humans
Hyperplasia
MAP Kinase Kinase 1
Muscle, Smooth, Vascular
Mutation
Myocytes, Smooth Muscle
Rabbits
ras Proteins
Signal Transduction
Simplexvirus
Tunica Intima
authors with profiles
Christopher Skelly
Ralph Weichselbaum