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Xu, Ming
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overview My long-term research interest is to understand mechanisms underlying drug addiction and to find possible treatment. We use behavioral, genetic, anatomical and molecular biological methods. One major research project is to identify mechanisms and neuronal circuits associated with reconsolidation and extinction of cocaine-induced reward memory. Another project is to develop a novel method of using skin stem cells to deliver therapeutic genes to combat the abuse of cocaine, ethanol, nicotine and their co-abuse.
One or more keywords matched the following items that are connected to Xu, Ming
Item TypeName
Concept Cocaine
Concept Cocaine-Related Disorders
Academic Article Dopamine D1 and D3 receptors are differentially involved in cue-elicited cocaine seeking.
Academic Article Dopamine D(1) and D(3) receptors oppositely regulate NMDA- and cocaine-induced MAPK signaling via NMDA receptor phosphorylation.
Academic Article Exploring mechanisms underlying extinction of cue-elicited cocaine seeking.
Academic Article Cocaine-induced intracellular signaling and gene expression are oppositely regulated by the dopamine D1 and D3 receptors.
Academic Article Dopamine D1 and D3 receptors mediate reconsolidation of cocaine memories in mouse models of drug self-administration.
Academic Article Repeated cocaine administration induces gene expression changes through the dopamine D1 receptors.
Academic Article Dopamine D3 receptors regulate reconsolidation of cocaine memory.
Academic Article c-Fos facilitates the acquisition and extinction of cocaine-induced persistent changes.
Academic Article Lack of self-administration of cocaine in dopamine D1 receptor knock-out mice.
Academic Article Activity-dependent modulation of limbic dopamine D3 receptors by CaMKII.
Academic Article Dopamine D1 and N-methyl-D-aspartate receptors and extracellular signal-regulated kinase mediate neuronal morphological changes induced by repeated cocaine administration.
Academic Article Activation of dopamine D3 receptors inhibits reward-related learning induced by cocaine.
Academic Article Genome-edited skin epidermal stem cells protect mice from cocaine-seeking behaviour and cocaine overdose
Academic Article Elimination of cocaine-induced hyperactivity and dopamine-mediated neurophysiological effects in dopamine D1 receptor mutant mice.
Academic Article Cellular responses to psychomotor stimulant and neuroleptic drugs are abnormal in mice lacking the D1 dopamine receptor.
Academic Article The dopamine D1 receptor is a critical mediator for cocaine-induced gene expression.
Academic Article Dopamine D3 receptor mutant mice exhibit increased behavioral sensitivity to concurrent stimulation of D1 and D2 receptors.
Academic Article Behavioral responses to cocaine and amphetamine administration in mice lacking the dopamine D1 receptor.
Academic Article Genome-edited skin epidermal stem cells protect mice from cocaine-seeking behaviour and cocaine overdose.
Academic Article An autophagy-related protein Becn2 regulates cocaine reward behaviors in the dopaminergic system.
Academic Article Reducing alcohol and/or cocaine-induced reward and toxicity via an epidermal stem cell-based gene delivery platform.
Grant LONG TERM COCAINE INDUCED CHANGES VIA DOPAMINE RECEPTORS
Grant Extinction of cue-elicited cocaine seeking
Grant Role of c-fos in cocaine actions
Grant A novel tool for studying D3 receptors and cocaine reward
Grant A cutaneous gene therapy for cocaine abuse and alcohol co-abuse
Grant Molecular Determinants of Compulsive Cocaine-Taking
Grant C-FOS AND PSYCHOMOTOR STIMULANT ACTIONS
Search Criteria
  • Cocaine