Transcription Factor CHOP
"Transcription Factor CHOP" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A CCAAT-enhancer binding protein that is induced by DNA DAMAGE and growth arrest. It serves as a dominant negative inhibitor of other CCAAT-enhancer binding proteins.
| Descriptor ID |
D051742
|
| MeSH Number(s) |
D12.776.260.108.124.875 D12.776.660.167.875 D12.776.930.127.124.875
|
| Concept/Terms |
Transcription Factor CHOP- Transcription Factor CHOP
- CHOP, Transcription Factor
- Growth Arrest And DNA Damage-Inducible Protein 153
- Growth Arrest And DNA Damage Inducible Protein 153
- DNA Damage-Inducible Transcript 3 Protein
- DNA Damage Inducible Transcript 3 Protein
- GADD153 Protein
- C-EBP Homologous Protein
- C EBP Homologous Protein
- Homologous Protein, C-EBP
- DDIT3 Protein
|
Below are MeSH descriptors whose meaning is more general than "Transcription Factor CHOP".
Below are MeSH descriptors whose meaning is more specific than "Transcription Factor CHOP".
This graph shows the total number of publications written about "Transcription Factor CHOP" by people in this website by year, and whether "Transcription Factor CHOP" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 1994 | 0 | 1 | 1 |
| 2000 | 0 | 1 | 1 |
| 2003 | 0 | 1 | 1 |
| 2007 | 0 | 1 | 1 |
| 2008 | 0 | 1 | 1 |
| 2010 | 0 | 2 | 2 |
| 2011 | 0 | 3 | 3 |
| 2016 | 0 | 1 | 1 |
| 2019 | 1 | 0 | 1 |
| 2021 | 0 | 1 | 1 |
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Below are the most recent publications written about "Transcription Factor CHOP" by people in Profiles.
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HRD1-mediated METTL14 degradation regulates m6A mRNA modification to suppress ER proteotoxic liver disease. Mol Cell. 2021 12 16; 81(24):5052-5065.e6.
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CHOP is dispensable for lens transparency in wild-type and connexin50 mutant mice. Mol Vis. 2019; 25:535-545.
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The UPR-PERK pathway is not a promising therapeutic target for mutant SOD1-induced ALS. Neurobiol Dis. 2019 07; 127:527-544.
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Activation of the Integrated Stress Response and Metabolic Dysfunction in a Murine Model of Sleep Apnea. Am J Respir Cell Mol Biol. 2017 10; 57(4):477-486.
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Integrated stress response activation by sleep fragmentation during late gestation in mice leads to emergence of adverse metabolic phenotype in offspring. Metabolism. 2017 04; 69:188-198.
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Intravenous Lipid Infusion Induces Endoplasmic Reticulum Stress in Endothelial Cells and Blood Mononuclear Cells of Healthy Adults. J Am Heart Assoc. 2016 Jan 11; 5(1).
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ER Chaperone BiP/GRP78 Is Required for Myelinating Cell Survival and Provides Protection during Experimental Autoimmune Encephalomyelitis. J Neurosci. 2015 Dec 02; 35(48):15921-33.
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Chronic sleep fragmentation during the sleep period induces hypothalamic endoplasmic reticulum stress and PTP1b-mediated leptin resistance in male mice. Sleep. 2015 Jan 01; 38(1):31-40.
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Oligodendrocyte-specific activation of PERK signaling protects mice against experimental autoimmune encephalomyelitis. J Neurosci. 2013 Apr 03; 33(14):5980-91.
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Endoplasmic reticulum stress decreases intracellular thyroid hormone activation via an eIF2a-mediated decrease in type 2 deiodinase synthesis. Mol Endocrinol. 2011 Dec; 25(12):2065-75.