"Receptors, Growth Factor" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Cell surface receptors that bind growth or trophic factors with high affinity, triggering intracellular responses which influence the growth, differentiation, or survival of cells.
| Descriptor ID |
D017978
|
| MeSH Number(s) |
D12.776.543.750.750.400
|
| Concept/Terms |
Receptors, Growth Factor- Receptors, Growth Factor
- Receptors, Growth Factors
- Growth Factors Receptors
- Growth Factor Receptors
Trophic Factor Receptors- Trophic Factor Receptors
- Receptors, Trophic Factor
- Trophic Factor Receptor
- Factor Receptor, Trophic
- Receptor, Trophic Factor
|
Below are MeSH descriptors whose meaning is more general than "Receptors, Growth Factor".
Below are MeSH descriptors whose meaning is more specific than "Receptors, Growth Factor".
This graph shows the total number of publications written about "Receptors, Growth Factor" by people in this website by year, and whether "Receptors, Growth Factor" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1994 | 1 | 0 | 1 |
| 1996 | 0 | 1 | 1 |
| 1997 | 0 | 2 | 2 |
| 1999 | 0 | 1 | 1 |
| 2000 | 0 | 1 | 1 |
| 2001 | 1 | 0 | 1 |
| 2002 | 0 | 1 | 1 |
| 2005 | 0 | 1 | 1 |
| 2008 | 1 | 1 | 2 |
| 2009 | 3 | 1 | 4 |
| 2010 | 1 | 3 | 4 |
| 2011 | 2 | 3 | 5 |
| 2019 | 0 | 1 | 1 |
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click here.
Below are the most recent publications written about "Receptors, Growth Factor" by people in Profiles.
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Growth factors with enhanced syndecan binding generate tonic signalling and promote tissue healing. Nat Biomed Eng. 2020 04; 4(4):463-475.
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RON (MST1R) is a novel prognostic marker and therapeutic target for gastroesophageal adenocarcinoma. Cancer Biol Ther. 2011 Jul 01; 12(1):9-46.
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The clathrin-binding domain of CALM-AF10 alters the phenotype of myeloid neoplasms in mice. Oncogene. 2012 Jan 26; 31(4):494-506.
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Activity of crizotinib (PF02341066), a dual mesenchymal-epithelial transition (MET) and anaplastic lymphoma kinase (ALK) inhibitor, in a non-small cell lung cancer patient with de novo MET amplification. J Thorac Oncol. 2011 May; 6(5):942-6.
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Personalized treatment of lung cancer. Semin Oncol. 2011 Apr; 38(2):274-83.
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Fyn is downstream of the HGF/MET signaling axis and affects cellular shape and tropism in PC3 cells. Clin Cancer Res. 2011 May 15; 17(10):3112-22.
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Rapid and dramatic radiographic and clinical response to an ALK inhibitor (crizotinib, PF02341066) in an ALK translocation-positive patient with non-small cell lung cancer. J Thorac Oncol. 2010 Dec; 5(12):2044-6.
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Anaplastic lymphoma kinase inhibition in non-small-cell lung cancer. N Engl J Med. 2010 Oct 28; 363(18):1693-703.
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Mutations in MUSK causing congenital myasthenic syndrome impair MuSK-Dok-7 interaction. Hum Mol Genet. 2010 Jun 15; 19(12):2370-9.
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CBL is frequently altered in lung cancers: its relationship to mutations in MET and EGFR tyrosine kinases. PLoS One. 2010 Jan 29; 5(1):e8972.