"Chemokine CX3CL1" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A CX3C chemokine that is a transmembrane protein found on the surface of cells. The soluble form of chemokine CX3CL1 can be released from cell surface by proteolysis and act as a chemoattractant that may be involved in the extravasation of leukocytes into inflamed tissues. The membrane form of the protein may also play a role in cell adhesion.
Descriptor ID |
D054428
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MeSH Number(s) |
D12.644.276.374.200.130.500 D12.776.467.374.200.130.500 D12.776.543.193 D23.125.300.130.500 D23.469.200.130.500 D23.529.374.200.130.500
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Concept/Terms |
Chemokine CX3CL1- Chemokine CX3CL1
- CX3CL1, Chemokine
- CX3CL1 Chemokine
- Chemokine, CX3CL1
- Neurotactin
- Chemokine (C-X3-C Motif) Ligand 1
- Fractalkine
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Below are MeSH descriptors whose meaning is more general than "Chemokine CX3CL1".
Below are MeSH descriptors whose meaning is more specific than "Chemokine CX3CL1".
This graph shows the total number of publications written about "Chemokine CX3CL1" by people in this website by year, and whether "Chemokine CX3CL1" was a major or minor topic of these publications.
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Year | Major Topic | Minor Topic | Total |
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2003 | 0 | 1 | 1 |
2010 | 1 | 0 | 1 |
2012 | 0 | 1 | 1 |
2024 | 1 | 0 | 1 |
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Below are the most recent publications written about "Chemokine CX3CL1" by people in Profiles.
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Mild Hypoxia Accelerates Cerebral Cavernous Malformation Disease Through CX3CR1-CX3CL1 Signaling. Arterioscler Thromb Vasc Biol. 2024 06; 44(6):1246-1264.
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Effects of small intestinal submucosa (SIS) on the murine innate immune microenvironment induced by heat-killed Staphylococcus aureus. PLoS One. 2012; 7(11):e48724.
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Fractalkine attenuates excito-neurotoxicity via microglial clearance of damaged neurons and antioxidant enzyme heme oxygenase-1 expression. J Biol Chem. 2011 Jan 21; 286(3):2308-19.
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Fractalkine (CX3CL1) stimulated by nuclear factor kappaB (NF-kappaB)-dependent inflammatory signals induces aortic smooth muscle cell proliferation through an autocrine pathway. Biochem J. 2003 Jul 15; 373(Pt 2):547-58.