S100/Calgranulin-mediated inflammation accelerates left ventricular hypertrophy and aortic valve sclerosis in chronic kidney disease in a receptor for advanced glycation end products-dependent manner.
S100/Calgranulin-mediated inflammation accelerates left ventricular hypertrophy and aortic valve sclerosis in chronic kidney disease in a receptor for advanced glycation end products-dependent manner. Arterioscler Thromb Vasc Biol. 2014 Jul; 34(7):1399-411.
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Animals
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Aortic Valve
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Calcinosis
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Calgranulin A
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Calgranulin B
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Cells, Cultured
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Diastole
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Disease Models, Animal
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Fibroblast Growth Factors
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Fibroblasts
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Genotype
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Heart Valve Diseases
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Humans
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Hypertrophy, Left Ventricular
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Inflammation
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Inflammation Mediators
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Leukocyte L1 Antigen Complex
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Mice
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Mice, Inbred C57BL
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Mice, Knockout
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Mice, Transgenic
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Myocardium
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Phenotype
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Receptor for Advanced Glycation End Products
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Receptors, Immunologic
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Renal Insufficiency, Chronic
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S100 Proteins
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S100A12 Protein
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Sclerosis
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Signal Transduction
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Time Factors
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Ventricular Dysfunction, Left
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Ventricular Function, Left
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Ventricular Remodeling