Nuclear Receptor Co-Repressor 1
"Nuclear Receptor Co-Repressor 1" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A nuclear protein that regulates the expression of genes involved in a diverse array of processes related to metabolism and reproduction. The protein contains three nuclear receptor interaction domains and three repressor domains and is closely-related in structure to NUCLEAR RECEPTOR CO-REPRESSOR 2.
| Descriptor ID |
D056971
|
| MeSH Number(s) |
D12.776.930.780.625.374
|
| Concept/Terms |
Nuclear Receptor Co-Repressor 1- Nuclear Receptor Co-Repressor 1
- Nuclear Receptor Co Repressor 1
- RIP140 Repressor
- Nuclear Factor RIP140
- Receptor Interacting Protein 140
- Retinoid X Receptor-Interacting Protein 13
- Retinoid X Receptor Interacting Protein 13
- Nuclear Receptor Co-Repressor RIP140
- Nuclear Receptor Co Repressor RIP140
|
Below are MeSH descriptors whose meaning is more general than "Nuclear Receptor Co-Repressor 1".
Below are MeSH descriptors whose meaning is more specific than "Nuclear Receptor Co-Repressor 1".
This graph shows the total number of publications written about "Nuclear Receptor Co-Repressor 1" by people in this website by year, and whether "Nuclear Receptor Co-Repressor 1" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 1998 | 0 | 2 | 2 |
| 2000 | 0 | 1 | 1 |
| 2001 | 0 | 1 | 1 |
| 2003 | 0 | 1 | 1 |
| 2005 | 0 | 2 | 2 |
| 2006 | 0 | 2 | 2 |
| 2007 | 0 | 1 | 1 |
| 2009 | 0 | 1 | 1 |
| 2010 | 0 | 1 | 1 |
| 2011 | 1 | 0 | 1 |
| 2014 | 1 | 0 | 1 |
| 2020 | 0 | 1 | 1 |
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Below are the most recent publications written about "Nuclear Receptor Co-Repressor 1" by people in Profiles.
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HDAC3 ensures stepwise epidermal stratification via NCoR/SMRT-reliant mechanisms independent of its histone deacetylase activity. Genes Dev. 2020 07 01; 34(13-14):973-988.
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NCoR1 and SMRT play unique roles in thyroid hormone action in vivo. Mol Cell Biol. 2015 Feb; 35(3):555-65.
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The nuclear receptor corepressor (NCoR) controls thyroid hormone sensitivity and the set point of the hypothalamic-pituitary-thyroid axis. Mol Endocrinol. 2011 Feb; 25(2):212-24.
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The effect of a PP2A inhibitor on the nuclear receptor corepressor pathway in glioma. J Neurosurg. 2010 Aug; 113(2):225-33.
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Retinoic acid signalling induces the differentiation of mouse fetal liver-derived hepatic progenitor cells. Liver Int. 2009 Nov; 29(10):1569-81.
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N-CoR pathway targeting induces glioblastoma derived cancer stem cell differentiation. Cell Cycle. 2007 Feb 15; 6(4):467-70.
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Regulation of nuclear coactivator and corepressor expression in mouse cerebellum by thyroid hormone. Thyroid. 2006 Mar; 16(3):211-6.
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A novel thyroid hormone receptor-beta mutation that fails to bind nuclear receptor corepressor in a patient as an apparent cause of severe, predominantly pituitary resistance to thyroid hormone. J Clin Endocrinol Metab. 2006 May; 91(5):1887-95.
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Negative regulation by thyroid hormone receptor requires an intact coactivator-binding surface. J Clin Invest. 2005 Sep; 115(9):2517-23.
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The nuclear receptor corepressors NCoR and SMRT decrease peroxisome proliferator-activated receptor gamma transcriptional activity and repress 3T3-L1 adipogenesis. J Biol Chem. 2005 Apr 08; 280(14):13600-5.