Nuclear Receptor Subfamily 1, Group F, Member 3
"Nuclear Receptor Subfamily 1, Group F, Member 3" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
An orphan nuclear receptor found in the THYMUS where it plays a role in regulating the development and maturation of thymocytes. An isoform of this protein, referred to as RORgammaT, is produced by an alternatively transcribed mRNA.
| Descriptor ID |
D057132
|
| MeSH Number(s) |
D12.776.260.643.182 D12.776.826.209.182
|
| Concept/Terms |
Nuclear Receptor Subfamily 1, Group F, Member 3- Nuclear Receptor Subfamily 1, Group F, Member 3
- Nuclear Receptor RZR-gamma
- Nuclear Receptor RZR gamma
- Receptor RZR-gamma, Nuclear
- ROR-gamma
- Orphan Nuclear Receptor ROR-gamma
- Orphan Nuclear Receptor ROR gamma
- RAR-related Orphan Receptor C
- RAR related Orphan Receptor C
- ROR-C Receptors
- ROR C Receptors
- Nuclear Receptor NR1F3
- Receptor NR1F3, Nuclear
- Orphan Nuclear Receptor NR1F3
|
Below are MeSH descriptors whose meaning is more general than "Nuclear Receptor Subfamily 1, Group F, Member 3".
Below are MeSH descriptors whose meaning is more specific than "Nuclear Receptor Subfamily 1, Group F, Member 3".
This graph shows the total number of publications written about "Nuclear Receptor Subfamily 1, Group F, Member 3" by people in this website by year, and whether "Nuclear Receptor Subfamily 1, Group F, Member 3" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2009 | 0 | 1 | 1 |
| 2011 | 0 | 1 | 1 |
| 2012 | 1 | 1 | 2 |
| 2014 | 0 | 2 | 2 |
| 2015 | 1 | 0 | 1 |
| 2020 | 0 | 1 | 1 |
| 2021 | 0 | 1 | 1 |
| 2023 | 0 | 1 | 1 |
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Below are the most recent publications written about "Nuclear Receptor Subfamily 1, Group F, Member 3" by people in Profiles.
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Commensal bacteria signal through TLR5 and AhR to improve barrier integrity and prevent allergic responses to food. Cell Rep. 2023 10 31; 42(10):113153.
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Wnt-ß-catenin activation epigenetically reprograms Treg cells in inflammatory bowel disease and dysplastic progression. Nat Immunol. 2021 04; 22(4):471-484.
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An Immunologic Mode of Multigenerational Transmission Governs a Gut Treg Setpoint. Cell. 2020 06 11; 181(6):1276-1290.e13.
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MUCOSAL IMMUNOLOGY. Individual intestinal symbionts induce a distinct population of ROR?? regulatory T cells. Science. 2015 Aug 28; 349(6251):993-7.
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Innate lymphoid cells facilitate NK cell development through a lymphotoxin-mediated stromal microenvironment. J Exp Med. 2014 Jun 30; 211(7):1421-31.
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ß-Catenin promotes colitis and colon cancer through imprinting of proinflammatory properties in T cells. Sci Transl Med. 2014 Feb 26; 6(225):225ra28.
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Induction of innate lymphoid cell-derived interleukin-22 by the transcription factor STAT3 mediates protection against intestinal infection. Immunity. 2014 Jan 16; 40(1):25-39.
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Expression of ROR?t marks a pathogenic regulatory T cell subset in human colon cancer. Sci Transl Med. 2012 Dec 12; 4(164):164ra159.
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Master regulators or lineage-specifying? Changing views on CD4+ T cell transcription factors. Nat Rev Immunol. 2012 11; 12(11):799-804.
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Transcription factor c-Maf mediates the TGF-ß-dependent suppression of IL-22 production in T(H)17 cells. Nat Immunol. 2011 Oct 16; 12(12):1238-45.