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Excess SMAD signaling contributes to heart and muscle dysfunction in muscular dystrophy.
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Excess SMAD signaling contributes to heart and muscle dysfunction in muscular dystrophy.
Excess SMAD signaling contributes to heart and muscle dysfunction in muscular dystrophy. Hum Mol Genet. 2014 Dec 20; 23(25):6722-31.
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PubMed
subject areas
Animals
Body Weight
Calcium-Binding Proteins
Disease Models, Animal
Gene Expression Regulation
Heart Function Tests
Humans
Latent TGF-beta Binding Proteins
Mice
Mice, Knockout
Muscle, Skeletal
Muscular Dystrophies
Mutation
Myocardium
Phosphorylation
Sarcoglycans
Signal Transduction
Smad2 Protein
Smad3 Protein
Smad4 Protein
Transforming Growth Factor beta