 Dopamine and cAMP-Regulated Phosphoprotein 32
"Dopamine and cAMP-Regulated Phosphoprotein 32" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A phosphoprotein that was initially identified as a major target of DOPAMINE activated ADENYLYL CYCLASE in the CORPUS STRIATUM. It regulates the activities of PROTEIN PHOSPHATASE-1 and PROTEIN KINASE A, and it is a key mediator of the biochemical, electrophysiological, transcriptional, and behavioral effects of DOPAMINE.
| Descriptor ID |
D051937
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| MeSH Number(s) |
D12.776.641.249 D12.776.744.459
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| Concept/Terms |
Dopamine and cAMP-Regulated Phosphoprotein 32- Dopamine and cAMP-Regulated Phosphoprotein 32
- Dopamine and cAMP Regulated Phosphoprotein 32
- Dopamine- and Cyclic AMP-Regulated Phosphoprotein Mr 32,000
- Dopamine and Cyclic AMP Regulated Phosphoprotein Mr 32,000
- Dopamine and cAMP-Regulated Phosphoprotein
- Dopamine and cAMP Regulated Phosphoprotein
- DARPP-32 Protein
- DARPP 32 Protein
- Dopamine and Adenosine 3',5'-Monophosphate-Regulated Phosphoprotein
- Dopamine and Adenosine 3',5' Monophosphate Regulated Phosphoprotein
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Below are MeSH descriptors whose meaning is more general than "Dopamine and cAMP-Regulated Phosphoprotein 32".
Below are MeSH descriptors whose meaning is more specific than "Dopamine and cAMP-Regulated Phosphoprotein 32".
This graph shows the total number of publications written about "Dopamine and cAMP-Regulated Phosphoprotein 32" by people in this website by year, and whether "Dopamine and cAMP-Regulated Phosphoprotein 32" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
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| 1989 | 0 | 1 | 1 | | 1995 | 0 | 1 | 1 | | 2000 | 0 | 1 | 1 | | 2002 | 0 | 1 | 1 | | 2006 | 0 | 1 | 1 | | 2008 | 0 | 1 | 1 | | 2011 | 0 | 1 | 1 |
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Below are the most recent publications written about "Dopamine and cAMP-Regulated Phosphoprotein 32" by people in Profiles.
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Catanzaro G, Pucci M, Viscomi MT, Lanuti M, Feole M, Angeletti S, Grasselli G, Mandolesi G, Bari M, Centonze D, D'Addario C, Maccarrone M. Epigenetic modifications of Dexras 1 along the nNOS pathway in an animal model of multiple sclerosis. J Neuroimmunol. 2016 05 15; 294:32-40.
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Singaraja RR, Huang K, Sanders SS, Milnerwood AJ, Hines R, Lerch JP, Franciosi S, Drisdel RC, Vaid K, Young FB, Doty C, Wan J, Bissada N, Henkelman RM, Green WN, Davis NG, Raymond LA, Hayden MR. Altered palmitoylation and neuropathological deficits in mice lacking HIP14. Hum Mol Genet. 2011 Oct 15; 20(20):3899-909.
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Bryant CD, Graham ME, Distler MG, Munoz MB, Li D, Vezina P, Sokoloff G, Palmer AA. A role for casein kinase 1 epsilon in the locomotor stimulant response to methamphetamine. Psychopharmacology (Berl). 2009 May; 203(4):703-11.
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Charvin D, Roze E, Perrin V, Deyts C, Betuing S, Pagès C, Régulier E, Luthi-Carter R, Brouillet E, Déglon N, Caboche J. Haloperidol protects striatal neurons from dysfunction induced by mutated huntingtin in vivo. Neurobiol Dis. 2008 Jan; 29(1):22-9.
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Veenstra-VanderWeele J, Qaadir A, Palmer AA, Cook EH, de Wit H. Association between the casein kinase 1 epsilon gene region and subjective response to D-amphetamine. Neuropsychopharmacology. 2006 May; 31(5):1056-63.
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Lovinger DM. NMDA receptors lose their inhibitions. Nat Neurosci. 2002 Jul; 5(7):614-6.
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Menei P, Pean JM, Nerrière-Daguin V, Jollivet C, Brachet P, Benoit JP. Intracerebral implantation of NGF-releasing biodegradable microspheres protects striatum against excitotoxic damage. Exp Neurol. 2000 Jan; 161(1):259-72.
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Wainwright MS, Perry BD, Won LA, O'Malley KL, Wang WY, Ehrlich ME, Heller A. Immortalized murine striatal neuronal cell lines expressing dopamine receptors and cholinergic properties. J Neurosci. 1995 Jan; 15(1 Pt 2):676-88.
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Won L, Price S, Wainer BH, Hoffmann PC, Bolam JP, Greengard P, Heller A. Correlated light and electron microscopic study of dopaminergic neurons and their synaptic junctions with DARPP-32-containing cells in three-dimensional reaggregate tissue culture. J Comp Neurol. 1989 Nov 01; 289(1):165-77.
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