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Expression and alternative splicing of folate pathway genes in HapMap lymphoblastoid cell lines.
Genetic inactivation of p62 leads to accumulation of hyperphosphorylated tau and neurodegeneration.
Hemorrhage or Ischemia? The Importance of 'Spotting' it Right.
Polygenic virulence factors involved in pathogenesis of 1997 Hong Kong H5N1 influenza viruses in mice.
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Genetic inactivation of p62 leads to accumulation of hyperphosphorylated tau and neurodegeneration.
Genetic inactivation of p62 leads to accumulation of hyperphosphorylated tau and neurodegeneration. J Neurochem. 2008 Jul; 106(1):107-20.
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PubMed
subject areas
Adaptor Proteins, Signal Transducing
Aging
Animals
Brain
Brain-Derived Neurotrophic Factor
Disease Models, Animal
Glycogen Synthase Kinase 3
Glycogen Synthase Kinase 3 beta
Heat-Shock Proteins
Humans
JNK Mitogen-Activated Protein Kinases
Metabolic Syndrome
Mice
Mice, Knockout
Mitogen-Activated Protein Kinase 1
Molecular Chaperones
Nerve Degeneration
Neurofibrillary Tangles
Neurons
Obesity
Phosphorylation
Proto-Oncogene Proteins c-akt
Sequestosome-1 Protein
Signal Transduction
Solubility
tau Proteins
authors with profiles
Susannah Spiess
Nancy Jean Cox