Serum Amyloid P-Component
"Serum Amyloid P-Component" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Amyloid P component is a small, non-fibrillar glycoprotein found in normal serum and in all amyloid deposits. It has a pentagonal (pentaxin) structure. It is an acute phase protein, modulates immunologic responses, inhibits ELASTASE, and has been suggested as an indicator of LIVER DISEASE.
Descriptor ID |
D000683
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MeSH Number(s) |
D12.776.049.407.875 D12.776.124.050.730 D12.776.395.690
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Concept/Terms |
Serum Amyloid P-Component- Serum Amyloid P-Component
- Amyloid P-Component, Serum
- P-Component, Serum Amyloid
- Serum Amyloid P Component
- Amyloid P Component
- P Component, Amyloid
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Below are MeSH descriptors whose meaning is more general than "Serum Amyloid P-Component".
Below are MeSH descriptors whose meaning is more specific than "Serum Amyloid P-Component".
This graph shows the total number of publications written about "Serum Amyloid P-Component" by people in this website by year, and whether "Serum Amyloid P-Component" was a major or minor topic of these publications.
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Year | Major Topic | Minor Topic | Total |
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1992 | 1 | 1 | 2 | 2005 | 1 | 0 | 1 | 2011 | 1 | 0 | 1 | 2012 | 1 | 0 | 1 | 2016 | 1 | 0 | 1 |
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Below are the most recent publications written about "Serum Amyloid P-Component" by people in Profiles.
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Balhara J, Shan L, Zhang J, Muhuri A, Halayko AJ, Almiski MS, Doeing D, McConville J, Matzuk MM, Gounni AS. Pentraxin 3 deletion aggravates allergic inflammation through a TH17-dominant phenotype and enhanced CD4 T-cell survival. J Allergy Clin Immunol. 2017 Mar; 139(3):950-963.e9.
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Kim J, Gozal D, Bhattacharjee R, Kheirandish-Gozal L. TREM-1 and pentraxin-3 plasma levels and their association with obstructive sleep apnea, obesity, and endothelial function in children. Sleep. 2013 Jun 01; 36(6):923-31.
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Diamond JM, Meyer NJ, Feng R, Rushefski M, Lederer DJ, Kawut SM, Lee JC, Cantu E, Shah RJ, Lama VN, Bhorade S, Crespo M, Demissie E, Sonett J, Wille K, Orens J, Weinacker A, Weill D, Arcasoy S, Shah PD, Belperio JA, Wilkes D, Ware LB, Palmer SM, Christie JD. Variation in PTX3 is associated with primary graft dysfunction after lung transplantation. Am J Respir Crit Care Med. 2012 Sep 15; 186(6):546-52.
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Diamond JM, Lederer DJ, Kawut SM, Lee J, Ahya VN, Bellamy S, Palmer SM, Lama VN, Bhorade S, Crespo M, Demissie E, Sonett J, Wille K, Orens J, Shah PD, Weinacker A, Weill D, Kohl BA, Deutschman CC, Arcasoy S, Shah AS, Belperio JA, Wilkes D, Reynolds JM, Ware LB, Christie JD. Elevated plasma long pentraxin-3 levels and primary graft dysfunction after lung transplantation for idiopathic pulmonary fibrosis. Am J Transplant. 2011 Nov; 11(11):2517-22.
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Zhang X, Jafari N, Barnes RB, Confino E, Milad M, Kazer RR. Studies of gene expression in human cumulus cells indicate pentraxin 3 as a possible marker for oocyte quality. Fertil Steril. 2005 Apr; 83 Suppl 1:1169-79.
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Spiegel DM, Sprague SM. Serum amyloid P component: a predictor of clinical beta 2-microglobulin amyloidosis. Am J Kidney Dis. 1992 May; 19(5):427-32.
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Spiegel DM, Costante N, Janiga AM, Haas M, Soltani K. Deposition and removal of cutaneous beta 2-microglobulin. Am J Nephrol. 1992; 12(5):330-5.
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