Chromosomal Proteins, Non-Histone
"Chromosomal Proteins, Non-Histone" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Nucleoproteins, which in contrast to HISTONES, are acid insoluble. They are involved in chromosomal functions; e.g. they bind selectively to DNA, stimulate transcription resulting in tissue-specific RNA synthesis and undergo specific changes in response to various hormones or phytomitogens.
| Descriptor ID |
D002868
|
| MeSH Number(s) |
D12.776.660.235 D12.776.664.235
|
| Concept/Terms |
Chromosomal Proteins, Non-Histone- Chromosomal Proteins, Non-Histone
- Proteins, Non-Histone Chromosomal
- Chromosomal Proteins, Nonhistone
- Nonhistone Chromosomal Proteins
- Chromosomal Proteins, Non Histone
- Non-Histone Chromosomal Proteins
- Non Histone Chromosomal Proteins
|
Below are MeSH descriptors whose meaning is more general than "Chromosomal Proteins, Non-Histone".
Below are MeSH descriptors whose meaning is more specific than "Chromosomal Proteins, Non-Histone".
This graph shows the total number of publications written about "Chromosomal Proteins, Non-Histone" by people in this website by year, and whether "Chromosomal Proteins, Non-Histone" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2000 | 1 | 0 | 1 |
| 2002 | 1 | 0 | 1 |
| 2003 | 2 | 2 | 4 |
| 2004 | 1 | 0 | 1 |
| 2005 | 2 | 0 | 2 |
| 2006 | 1 | 0 | 1 |
| 2008 | 2 | 0 | 2 |
| 2009 | 4 | 1 | 5 |
| 2010 | 0 | 1 | 1 |
| 2012 | 2 | 2 | 4 |
| 2013 | 1 | 0 | 1 |
| 2014 | 3 | 3 | 6 |
| 2015 | 5 | 0 | 5 |
| 2016 | 2 | 0 | 2 |
| 2017 | 1 | 2 | 3 |
| 2018 | 2 | 0 | 2 |
| 2019 | 2 | 2 | 4 |
| 2020 | 1 | 1 | 2 |
| 2022 | 3 | 0 | 3 |
| 2024 | 0 | 1 | 1 |
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Below are the most recent publications written about "Chromosomal Proteins, Non-Histone" by people in Profiles.
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PU.1 and BCL11B sequentially cooperate with RUNX1 to anchor mSWI/SNF to poise the T cell effector landscape. Nat Immunol. 2024 May; 25(5):860-872.
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Synthetic Antibodies Detect Distinct Cellular States of Chromosome Passenger Complex Proteins. J Mol Biol. 2022 06 30; 434(12):167602.
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The three-dimensional structure of Epstein-Barr virus genome varies by latency type and is regulated by PARP1 enzymatic activity. Nat Commun. 2022 01 17; 13(1):187.
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The SWI/SNF chromatin remodeling assemblies BAF and PBAF differentially regulate cell cycle exit and cellular invasion in vivo. PLoS Genet. 2022 01; 18(1):e1009981.
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SWI/SNF Complex Mutations Promote Thyroid Tumor Progression and Insensitivity to Redifferentiation Therapies. Cancer Discov. 2021 05; 11(5):1158-1175.
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Uncoupling histone H3K4 trimethylation from developmental gene expression via an equilibrium of COMPASS, Polycomb and DNA methylation. Nat Genet. 2020 06; 52(6):615-625.
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A mutant form of Dmc1 that bypasses the requirement for accessory protein Mei5-Sae3 reveals independent activities of Mei5-Sae3 and Rad51 in Dmc1 filament stability. PLoS Genet. 2019 12; 15(12):e1008217.
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A Pliable Mediator Acts as a Functional Rather Than an Architectural Bridge between Promoters and Enhancers. Cell. 2019 08 22; 178(5):1145-1158.e20.
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A human postcatalytic spliceosome structure reveals essential roles of metazoan factors for exon ligation. Science. 2019 02 15; 363(6428):710-714.
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RPA resolves conflicting activities of accessory proteins during reconstitution of Dmc1-mediated meiotic recombination. Nucleic Acids Res. 2019 01 25; 47(2):747-761.